Publications

• Chiao JW. "What causes prostate cancer-learning from a tragedy." Carcinogenesis, 42(10), (2021) 1221-1222. doi: 10.1093/carcin/bgab078
• Wu J, Cang S, Liu C, et. al. "Development of human prostate cancer stem cells involves epigenomic alteration and PI3K/AKT pathway activation." Experimental hematology & oncology, 9(), (2020) 12. doi: 10.1186/s40164-020-00168-0
• Chen Y, Cang S, Han L, et. al. "Establishment of prostate cancer spheres from a prostate cancer cell line after phenethyl isothiocyanate treatment and discovery of androgen-dependent reversible differentiation between sphere and neuroendocrine cells." Oncotarget, 7(18), (2016) 26567-79. doi: 10.18632/oncotarget.8440
• Cang S, Xu X, Ma Y, et. al. "Hypoacetylation, hypomethylation, and dephosphorylation of H2B histones and excessive histone deacetylase activity in DU-145 prostate cancer cells." Journal of hematology & oncology, 9(), (2016) 3. doi: 10.1186/s13045-016-0233-x
• Chiao JW, Melikian M, Han L, et. al. "Interaction of a small molecule Natura-α and STAT3-SH2 domain to block Y705 phosphorylation and inhibit lupus nephritis." Biochemical pharmacology, 99(), (2016) 123-31. doi: 10.1016/j.bcp.2015.11.018
• Xue C, Pasolli HA, Piscopo I, et. al. "Mitochondrial structure alteration in human prostate cancer cells upon initial interaction with a chemopreventive agent phenethyl isothiocyanate." Cancer cell international, 14(1), (2014) 30. doi: 10.1186/1475-2867-14-30
• Cang S, Ma Y, Chiao JW, et. al. "Phenethyl isothiocyanate and paclitaxel synergistically enhanced apoptosis and alpha-tubulin hyperacetylation in breast cancer cells." Experimental hematology & oncology, 3(1), (2014) 5. doi: 10.1186/2162-3619-3-5

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